TLR7/TLR8 ACTIVATION AND SUSCEPTIBILITY GENES SYNERGIZE TO BREACH GUT BARRIER IN A MOUSE MODEL OF LUPUS



Molecular Network Analysis Suggests a Logical Hypothesis for the Pathological Role of C9orf72 in Amyotrophic Lateral Sclerosis/Frontotemporal Dementia

Background Expanded GGGGCC hexanucleotide repeats, ranging from hundreds to thousands in number, located in the noncoding region of the chromosome 9 open reading frame 72 ( C9orf72 ) gene represent the most common genetic abnormality for familial LCM Wire and sporadic amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) (abbreviate

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